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Pathways and mechanisms that control oxidative stress such as GSH metabolism, can modulate pathology and cognitive deficits in rodent models of schizophrenia (Cabuncgal et al

Several lines of evidence suggest decreased oxidative metabolism in some brain areas in SZ (Yao et al., 2004a

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For these experiments, cells were first treated with 50 g/mL and 100 g/mL of the protein synthesis inhibitors cycloheximide and chloramphenicol, respectively, for defined periods